Endocrine Disruptors
Organizational and Activational Effects

What is an Endocrine Disruptor?
Natural chemical (phytochemical)
Synthetic chemical (xenobiotic)
Enters body
Disrupts normal endocrine events

Where are EDs Found?
Plants:
Human-made
Plastics
Drugs
Household products
Industrial chemicals
Pesticides

Slide 4
Slide 5
Mechanisms of Action
Bind steroid hormone receptors and mimic
Block hormone binding
Alter cell-signaling & gene expression without binding
Influence production or metabolism of hormones
Influence hormone receptor production or action
Influence enzyme-related hormone functions

Most EDs
Estrogenic
Androgenic
Anti-androgenic

Slide 8
Slide 9
Slide 10
Slide 11
Cell Signaling
Not fully understood
DDT
Estrogenic effects--unknown pathway
Direct binding with androgen receptors

Hypothalamic-Pituitary-Gonadal
Axis
Organizational and Activational Hypothesis
Reproductive organs are organized and formed during the larval period with the help of steroid hormones.
These organs are later activated during the organismÕs reproductive period by the same hormones that organized these structures.

Slide 15
About Steroid HormonesÉ
Small amounts = potent effects
Travel with carrier proteins
Bind cytoplasmic or nuclear receptors
Exert genomic effects
Organization and Activation

How are EDs different or similar?
A little goes a long way
­ bioavailability b/c no need for carrier
Can exert more than one effect
Hand-me-down toxin

ED Target Tissues
Organs with gonadal hormone receptors
Females = mammary glands, reproductive tract
Males = reproductive tract
Both sexes = external genitalia, brain, skeleton, thyroid, liver, kidney, and immune system

Slide 19
Slide 20
Slide 21
Slide 22
Effects
Permanent if organizational
Morphological
Behavioral
Immunological
ø Stress response

The ED Timeline
A ÒSilentÓ Omen
Rachel Carson
1907-1964
Silent Spring (1962)
Over-use of pesticides
DDT (1945)
DDT ban 1972

Slide 26
Slide 27
Theo Colborn
WWF scientist
BS 1947 Rutgers
PhD 1985 UW Mad.
Studied Great Lakes
1991 EDs
Our Stolen Future 1996
Combines human and animal research on EDs
Explosion of ED research

Reptiles and Amphibians
Slide 30
Slide 31
Slide 32
Amphibian Work
Reeder et al. 1998, Rania 1998
ÒÉhalf the manÓ
Lou Guillette 1993
Lake Apopka and Tower Chemical 1980
Alligator infertility 1994

Lake Apopka Gators
Circulating sex hormone levels with RIA
Females = low E2
Males = low T
High E2 in testes
Pesticides with GC

Water Snakes and Coal Ash
EDs in prey items
Affect liver
Hopkins (2000)
Ganser et al. (2003)
Trace elements
Selenium
Arsenic
(Mercury)

Slide 38
Slide 39
Slide 40
Slide 41
Slide 42
Organizational Effects
Most prevalent
Permanent
Larvae and adults
Affect morphology
Behavior (shore birds)
Reproductive success
Populations

Activational Effects
Affect receptor populations
Affect receptor binding

Slide 45
Study Points Not Limited ToÉ
Types (classes) of hormones
Endocrine vs. Exocrine Glands
Feedback Loops
Interaction between hypothalamus and pituitary gland
Specific hormonal problems--be able to identify feedback anomalies
Disruptors vs. Natural Hormones
Organizational Activational Hypothesis

Be a Physiological Detective
Pinpoint problem area in feedback loop
Predict effects
What symptoms might you look for?
See if you can solve the following problems:

Thyroid Storm
Patient presents with:
Hypermetabolism
Fever
Rapid heart rate
Irritability
Exopthalmia
Serum TH levels are high
Where is the problem in the feedback loop?
Which organ would you treat to stop the loop?

Birthday
A pregnant woman surpasses her due date.
She presents at the emergency room without any physical symptoms besides a decreased amount of fetal movement.
Though the baby is not too distressed, you feel you must deliver the baby
What can you do?

Swollen Neck Glands
Your patient is a visitor from a different country.
She presents with a palpable mass lateral to her trachea.
She complains of feeling cold and sluggish
In her patient history, you find that her diet is low in iodine.
Where is the problem in the feedback loop?

Slide 51
Slide 52